Redhe O & Pleva J. Recovery from amyotrophic lateral sclerosis and from allergy after removal of dental amalgam fillings. Int J Risk & Safety in Med 4:229-236 (1994)

No abstract available. Citation from the text follows: "...Five months after the completion of DA removal (29 August 1984) the patient was called for a week-long investigation at the same University clinic where the diagnosis ALS had been made. She felt now extraordinarily healthy and her health status was also confirmed by the words in her record:"The neurologic status is completely without comment. Hence, the patient does not show any motor neuron disease of type ALS. She has been informed that she is in neurological respect fully healthy." ...At the time of writing (early 1993), 9 years have elapsed since removal of the DA fillings, and the patient continues to enjoy good health... "

Wenstrup D; Ehmann WD; Markesbery WR; Trace element imbalances in isolated subcellular fractions of Alzheimer's disease brains. Department of Chemistry, University of Kentucky, Lexington. Brain Res, 533: 1, 1990 Nov 12, 125-31

Concentrations of 13 trace elements (Ag, Br, Co, Cr, Cs, Fe, Hg, K, Na, Rb, Sc, Se, Zn) in isolated subcellular fractions (whole brain, nuclei, mitochondria, microsomes) of temporal lobe from autopsied Alzheimer's disease (AD) patients and normal controls were determined utilizing instrumental neutron activation analysis. Comparison of AD and controls revealed elevated Br (whole brain) and Hg (microsomes) and diminished Rb (whole brain, nuclear and microsomes), Se (microsomes) and Zn (nuclear) in AD. The elevated Br and Hg and diminished Rb are consistent with our previous studies in AD bulk brain specimens. Comparison of element ratios revealed increased Hg/Se, Hg/Zn and Zn/Se mass ratios in AD. Se and Zn play a protective role against Hg toxicity and our data suggest that they are utilized to detoxify Hg in the AD brain. Overall our studies suggest that Hg could be an important toxic element in AD. Whether Hg deposition in AD is a primary or secondary event remains to be determined.

Thompson CM; Markesbery WR; Ehmann WD; Mao YX; Vance DE; Regional brain trace-element studies in Alzheimer's disease. Department of Chemistry, University of Kentucky, Lexington 40506. Neurotoxicology, 9: 1, 1988 Spring, 1-7

Alzheimer's disease (AD) brain trace-element imbalances in the amygdala, hippocampus and nucleus basalis of Meynert (nbM) are found in most cases to be consistent with those previously reported in samples derived principally from AD cerebral cortex (Ehmann et al., 1986). The elevation of mercury in AD nbM, as compared to age-matched controls, is the largest trace-element imbalance observed to date in AD brain. In addition to the general confirmation of imbalances for Cs, Hg, N, Na, P, and Rb noted previously in cerebral cortex samples, imbalances for Fe, K, Sc, and Zn were observed in two regions and one region also exhibited imbalances for both Co and Se. Persistent imbalances for the univalent cations Na, K, Rb and Cs support arguments for a membrane abnormality in AD. The data presented here also provide the first comprehensive simultaneous multi-element determinations in both control and AD nbM.

Also make sure to read these books: Poison in Your Teeth: Mercury Amalgam (Silver) Fillings...Hazardous to Your Health! and Mercury Detoxification by Tom McGuire